Murray, J., Craggs, G., Wilson, L., & Kellie, S. (2000). Mechanism of phosphatidylinositol 3-kinase-dependent increases in BAC1.2F5 macrophage-like cell density in response to M-CSF: Phosphatidylinositol 3-kinase inhibitors increase the rate of apoptosis rather than inhibit DNA synthesis. Inflammation Research, 49(11), pp. 610-618. doi:10.1007/s000110050638
Chicago Style CitationMurray, James, G. Craggs, L. Wilson, and S. Kellie. "Mechanism of Phosphatidylinositol 3-kinase-dependent Increases in BAC1.2F5 Macrophage-like Cell Density in Response to M-CSF: Phosphatidylinositol 3-kinase Inhibitors Increase the Rate of Apoptosis Rather Than Inhibit DNA Synthesis." Inflammation Research 49, no. 11 (2000): 610-618.
MLA CitationMurray, James, G. Craggs, L. Wilson, and S. Kellie. "Mechanism of Phosphatidylinositol 3-kinase-dependent Increases in BAC1.2F5 Macrophage-like Cell Density in Response to M-CSF: Phosphatidylinositol 3-kinase Inhibitors Increase the Rate of Apoptosis Rather Than Inhibit DNA Synthesis." Inflammation Research 49.11 (2000): 610-618.